A review about biomarkers for the investigation of vascular function and impairment in diabetes mellitus

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Term Occurence Count Dictionary
hyperhomocysteinemia 1 endocrinologydiseases
hyperinsulinemia 2 endocrinologydiseases
hyperlipidemia 1 endocrinologydiseases
hypertriglyceridemia 2 endocrinologydiseases
obesity 1 endocrinologydiseases
type 2 diabetes mellitus 3 endocrinologydiseases
Insulin 1 endocrinologydiseasesdrugs
diabetes mellitus 5 endocrinologydiseases

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Select Drug Character Offset Drug Term Instance
Insulin 4301 consequent synthesis and release of cytokines, vasoadhesion molecules, endothelin-1, and tissue factor. Insulin resistance and hyperinsulinemiaUnder physiologic conditions, other than the hypoglycemic function, insulin
Select Disease Character Offset Disease Term Instance
diabetes mellitus 129 Risk ManagementA review about biomarkers for the investigation of vascular function and impairment in diabetes mellitus Giuseppe DerosaPamela Maffioli1Centre of Diabetes and Metabolic Diseases, Department of Internal Medicine
diabetes mellitus 972 (high-sensitivity C-reactive protein, interleukin-6, and tumor necrosis factor-α) were elevated in type 2 diabetes mellitus . Their increased expression and release contribute to the accelerated atherogenesis typical of these
diabetes mellitus 1276 biomarkers will help to establish new strategies to reduce cardiovascular complications.IntroductionType 2 diabetes mellitus is responsible for high mortality rates, approximately twice that of the general population: micro-
diabetes mellitus 2539 elevated free fatty acids), and hyperhomocysteinemia are important pathophysiological components of type 2 diabetes mellitus that trigger systemic inflammation and impair nitric oxide (NO) bioavailability, with consequent impaired
diabetes mellitus 13752 sE-selectin) and inflammatory cytokines (high-sensitivity CRP, IL-6, and TNF-α) are elevated in type 2 diabetes mellitus . Their increased expression and release contribute to the accelerated atherogenesis typical of these
hyperhomocysteinemia 2464 insulin resistance, hyperinsulinemia, hyperlipidemia (in particular elevated free fatty acids), and hyperhomocysteinemia are important pathophysiological components of type 2 diabetes mellitus that trigger systemic inflammation
hyperinsulinemia 2384 infarction, stroke, angina pectoris, or peripheral artery disease.[5],[6] Hyperglycemia, insulin resistance, hyperinsulinemia , hyperlipidemia (in particular elevated free fatty acids), and hyperhomocysteinemia are important pathophysiological
hyperinsulinemia 4324 release of cytokines, vasoadhesion molecules, endothelin-1, and tissue factor.Insulin resistance and hyperinsulinemia Under physiologic conditions, other than the hypoglycemic function, insulin has also a hemodynamic action
hyperlipidemia 2402 pectoris, or peripheral artery disease.[5],[6] Hyperglycemia, insulin resistance, hyperinsulinemia, hyperlipidemia (in particular elevated free fatty acids), and hyperhomocysteinemia are important pathophysiological
hypertriglyceridemia 5107 role in the endothelial damage. We have already demonstrated in two previous studies we conducted that hypertriglyceridemia , in particular postprandial hypertriglyceridemia simulated by an oral fat load, is responsible for an
hypertriglyceridemia 5156 demonstrated in two previous studies we conducted that hypertriglyceridemia, in particular postprandial hypertriglyceridemia simulated by an oral fat load, is responsible for an elevated inflammatory state with an increase in
obesity 13353 arteries and aneurysm formation in animals and humans.[47] MMP-2 and MMP-9 are elevated in patients with obesity ,[48] hypertension,[49] type 2 diabetes,[50] and acute coronary syndrome.[51]–[53] Moreover, plasma
type 2 diabetes mellitus 965 (high-sensitivity C-reactive protein, interleukin-6, and tumor necrosis factor-α) were elevated in type 2 diabetes mellitus . Their increased expression and release contribute to the accelerated atherogenesis typical of these
type 2 diabetes mellitus 2532 elevated free fatty acids), and hyperhomocysteinemia are important pathophysiological components of type 2 diabetes mellitus that trigger systemic inflammation and impair nitric oxide (NO) bioavailability, with consequent impaired
type 2 diabetes mellitus 13745 and sE-selectin) and inflammatory cytokines (high-sensitivity CRP, IL-6, and TNF-α) are elevated in type 2 diabetes mellitus . Their increased expression and release contribute to the accelerated atherogenesis typical of these

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