Renal function monitoring in heart failure - what is the optimal frequency? A narrative review.

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Term Occurence Count Dictionary
kidney failure 1 nephrologydiseases
bumetanide 1 nephrologydiseasesdrugs
chlorothiazide 2 nephrologydiseasesdrugs
chronic kidney disease 2 nephrologydiseases
interstitial nephritis 1 nephrologydiseases
acute kidney injury 2 nephrologydiseases
furosemide 2 nephrologydiseasesdrugs
glomerulonephritis 1 nephrologydiseases
hydrochlorothiazide 1 nephrologydiseasesdrugs

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Select Drug Character Offset Drug Term Instance
bumetanide 37287 observed pharmacological effect (Table 4). Studies looking at the timed response to the loop diuretic bumetanide have shown that its diuretic effect occurs <1 h after oral administration. Maximal effect is achieved
chlorothiazide 37809 with the same dose given repeatedly 76. A similar effect has been seen with the thiazide diuretic hydro chlorothiazide 77. The greatest diuretic effect is seen with the first few doses, causing significant electrolyte shifts
chlorothiazide 38565 diureticsMaximum daily doseDuration of actionChlorothiazide1000 mg6–12 hChlorthalidone100 mg24–72 hHydro chlorothiazide 200 mg6–12 hIndapamide5 mg36 hMetolazone20 mg12–24 hThese observations have implications for
furosemide 32969 failure (October 2013) 68A pragmatic approach to diuretic prescribing is described. It is suggested that furosemide , the most common diuretic in heart failure, is titrated up based on daily weight of the patient to achieve
furosemide 37609 for the same concentration) 75. These findings are supported by observed actions of the loop diuretic furosemide , which shows a maximal effect within 1.5 h of the first oral dose and reduced effect with the same
hydrochlorothiazide 37804 effect with the same dose given repeatedly 76. A similar effect has been seen with the thiazide diuretic hydrochlorothiazide 77. The greatest diuretic effect is seen with the first few doses, causing significant electrolyte shifts
Select Disease Character Offset Disease Term Instance
acute kidney injury 2435 5–17. doi: 10.1111/bcp.13434.28901643What is Already Known about this SubjectDiuretics can lead to acute kidney injury , which is one of the most common causes of hospital admission due to adverse drug reactions.There have
acute kidney injury 5905 of which reflects the presumed primary and secondary problem 17: Type 1: Acute heart failure causes acute kidney injury (AKI)Type 2: Chronic heart failure causes CKDType 3: AKI or acute renal failure causes acute cardiac
chronic kidney disease 3730 hospital admission and deterioration in these patients is worsening renal function 3. Prevalence of chronic kidney disease (CKD) ranges from 39 to 60% in heart failure cohorts and is associated with increased mortality and
chronic kidney disease 32764 Association; ACEi, angiotensin‐converting enzyme inhibitor; ARB, angiotensin receptor blockers; CKD, chronic kidney disease ; WRF, worsening renal functionACCF/AHA guideline for the management of heart failure (October 2013)
glomerulonephritis 46412 histological changes, or it might result (albeit rarely) from an intrinsic renal pathology such as glomerulonephritis . Clearly the clinician needs to apply clinical context and common sense to interpretation of the creatinine
interstitial nephritis 13989 because of its antiplatelet effects. Rarely, aspirin can cause an idiosyncratic reaction causing tubulo‐ interstitial nephritis , which can lead to AKI. This is rare at low doses of 75 mg, although the risk is slightly higher if
kidney failure 20553 monitoring can help clinicians intervene at a stage sufficiently early to reduce risk of progression to kidney failure .Frequency of renal monitoringNo study has specifically assessed frequency and optimal timing of renal

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