Septic acute kidney injury: molecular mechanisms and the importance of stratification and targeting therapy.

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acute kidney injury 4 nephrologydiseases
kidney failure 2 nephrologydiseases

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acute kidney injury 27 Title: Critical CareSeptic acute kidney injury : molecular mechanisms and the importance of stratification and targeting therapyEric D MorrellJohn A
acute kidney injury 324 9/2014Publication date (pmc-release): 9/2014Publication date (ppub): /2014AbstractThe most common cause of acute kidney injury (AKI) in hospitalized patients is sepsis. However, the molecular pathways and mechanisms that mediate
acute kidney injury 3780 manifestations in septic AKI occur prior to significant cell death secondary to apoptosis or necrosis.Septic acute kidney injury : is it hyperemic kidney injury?The morphologic changes that are seen in classic ischemic and toxic acute
acute kidney injury 22733 differences in the two species’ genomic responses to stress [[77]].Does apoptosis have a role in septic acute kidney injury ?The traditional model of apoptosis in kidney and other cells is similar [[78]]. Processes involving
kidney failure 11911 days after treatment in patients treated with the agent than in those not treated. Although rates of kidney failure were not reported in this trial, there was a statistically significant improvement in the overall Sequential
kidney failure 23340 necrosis factor receptor-1 (TNFR-1; Figure 2). TNFR-1 knockout mice were resistant to LPS-induced kidney failure while they also manifested much less apoptosis and neutrophil accumulation, suggesting a direct role

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