Hypoxia, HIF, and Associated Signaling Networks in Chronic Kidney Disease.

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Term Occurence Count Dictionary
acute kidney injury 1 nephrologydiseases
chronic kidney disease 2 nephrologydiseases
focal segmental glomerulosclerosis 1 nephrologydiseases
glomerulonephritis 1 nephrologydiseases

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acute kidney injury 8013 controversial whether HIF is pro-fibrotic or anti-fibrotic. To define the role of HIF activation on ischemic acute kidney injury (AKI) associated fibrosis, Kapitsinou P et al. inhibited PHD by pharmacological inhibitor to increase
chronic kidney disease 740 (G.D.)Publication date (epub): 4/2017Publication date (collection): 5/2017AbstractThe pathogenesis of chronic kidney disease (CKD) is complex and apparently multifactorial. Hypoxia or decrease in oxygen supply in kidney tissues
chronic kidney disease 17764 significantly enhances Notch3 expression [[63]]. In addition, in angiotensin II infusion model of hypertensive chronic kidney disease , HIF-1α has been shown to be essential to initiate the glomerular injury and progression to renal fibrosis
focal segmental glomerulosclerosis 13987 matrix remodeling and deposition (Figure 1). In anti-Tac(Fv)-PE38(LMB2)-induced podocyte injury and focal segmental glomerulosclerosis model, HIF-1α has been reported to increase and bind to the HRE of collagen type I alpha 2 chain (COL1A2)
glomerulonephritis 2209 economic and health burden on both the affected families and the whole society.Diabetes, hypertension and glomerulonephritis are the leading causes of CKD and multiple underlying mechanisms have been suggested. Among them, hypoxia

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