Acute kidney injury: current concepts and new insights

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Annotation Summary

Term Occurence Count Dictionary
interstitial nephritis 3 nephrologydiseases
metabolic acidosis 1 nephrologydiseases
Contrast-induced nephropathy 1 nephrologydiseases
acute kidney injury 10 nephrologydiseases
bumetanide 1 nephrologydiseasesdrugs
chronic kidney disease 3 nephrologydiseases
furosemide 1 nephrologydiseasesdrugs
glomerulonephritis 2 nephrologydiseases

Graph of close proximity drug and disease terms (within 200 characters).

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Select Drug Character Offset Drug Term Instance
bumetanide 12250 use of this strategy is not recommended.[17],[32],[33]Although loop diuretics such as furosemide and bumetanide are commonly used in the management of AKI, their use are not recommended for the prevention or treatment
furosemide 12235 systematic reviews, use of this strategy is not recommended.[17],[32],[33]Although loop diuretics such as furosemide and bumetanide are commonly used in the management of AKI, their use are not recommended for the prevention
Select Disease Character Offset Disease Term Instance
Contrast-induced nephropathy 8580 of AKI is the use of iodinated contrast agents in the diagnostic procedures such as angiography.[20] Contrast-induced nephropathy can be prevented by use of iso-osmolar agents and isotonic saline infusion.[20],[21]Novel BiomarkersAs
acute kidney injury 518 frequently seen in hospitalized patients, especially in critically ill patients. The primary causes of acute kidney injury are divided into three categories: prerenal, intrinsic renal and postrenal. The definition and staging
acute kidney injury 644 divided into three categories: prerenal, intrinsic renal and postrenal. The definition and staging of acute kidney injury are mainly based on the risk, injury, failure, loss, end-stage kidney disease (RIFLE) criteria and the
acute kidney injury 767 mainly based on the risk, injury, failure, loss, end-stage kidney disease (RIFLE) criteria and the acute kidney injury network (AKIN) criteria, which have previously been defined. However the clinical utility of these criteria
acute kidney injury 1116 suggested for the diagnosis, severity classification and most importantly, the modification of outcome in acute kidney injury .Methods:Current literature on the definition, biomarkers, management and epidemiology of acute kidney
acute kidney injury 1225 kidney injury.Methods:Current literature on the definition, biomarkers, management and epidemiology of acute kidney injury was reviewed by searching keywords in Medline and PubMed databases.Results:The epidemiology, pathophysiology
acute kidney injury 1371 keywords in Medline and PubMed databases.Results:The epidemiology, pathophysiology and diagnosis of acute kidney injury were discussed. The clinical implications of novel biomarkers and management of acute kidney injury
acute kidney injury 1471 acute kidney injury were discussed. The clinical implications of novel biomarkers and management of acute kidney injury were also discussed.Conclusions:The current definitions of acute kidney injury are based on the RIFLE,
acute kidney injury 1550 biomarkers and management of acute kidney injury were also discussed.Conclusions:The current definitions of acute kidney injury are based on the RIFLE, AKIN and KDIGO criteria. Although these criteria have been widely validated,
acute kidney injury 1715 criteria. Although these criteria have been widely validated, some of limitations are still remain. Since acute kidney injury is common and harmful, all preventive measures should be taken to avoid its occurrence. Currently, there
acute kidney injury 3243 are based on the risk, injury, failure, loss, end-stage kidney disease (RIFLE) criteria [4] and the acute kidney injury network (AKIN) criteria,[5] which have previously been defined. The incidence of AKI varies according
chronic kidney disease 5903 patients with increased sCr concentrations, it is important to distinguish whether the patient has AKI, chronic kidney disease , or a bout of acute illness superimposed on a chronic disease. In this context, some diagnostic clues
chronic kidney disease 6057 superimposed on a chronic disease. In this context, some diagnostic clues that suggest the presence of chronic kidney disease may be helpful, namely, abnormal sCr prior to presentation, associated risk factors (eg, hypertension
chronic kidney disease 9768 considered a superior marker when compared with sCr in both animal models and clinical settings of chronic kidney disease .[26],[27] However, it is unclear whether the value of cystatin C is generarizable to all forms of AKI
glomerulonephritis 6444 kidneys.[16]In some cases, AKI occurs secondary to inflammatory parenchymal diseases such as vasculitis, glomerulonephritis and interstitial nephritis. In such patients, the clinical features of these diagnoses including systemic
glomerulonephritis 6641 diagnoses including systemic manifestations in vasculitis, the presence of macroscopic haematuria in glomerulonephritis and/or the recent initiation of treatment with a drug known to cause interstitial nephritis should be
interstitial nephritis 4337 syndrome encompassing various etiologies, including acute tubular necrosis, pre-renal azotemia, acute interstitial nephritis , acute glomerular and vasculitic renal diseases, and acute postrenal obstructive nephropathy. Some of
interstitial nephritis 6467 AKI occurs secondary to inflammatory parenchymal diseases such as vasculitis, glomerulonephritis and interstitial nephritis . In such patients, the clinical features of these diagnoses including systemic manifestations in vasculitis,
interstitial nephritis 6729 haematuria in glomerulonephritis and/or the recent initiation of treatment with a drug known to cause interstitial nephritis should be considered. Malignant hypertension, bilateral cortical necrosis, pyelonephritis, amyloidosis
metabolic acidosis 12922 oliguria (urine output<200 ml over 12h)3. Hyperkalemia (potassium concentration >6.5 mmol/L)4. Severe metabolic acidosis (pH<7.2 despite normal or low partial pressure of carbon dioxide in arterial blood)5. Volume overload

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