Anti-NR2A/B Antibodies and Other Major Molecular Mechanisms in the Pathogenesis of Cognitive Dysfunction in Systemic Lupus Erythematosus

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autoimmune disease 3 rheumatologydiseases
systemic lupus erythematosus 2 rheumatologydiseases

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autoimmune disease 724 (epub): 5/2015Publication date (collection): 5/2015AbstractSystemic lupus erythematosus (SLE) is an autoimmune disease that affects approximately 1–45.3 per 100,000 people worldwide. Although deaths as a result of active
autoimmune disease 2334 cognitive decline in SLE patients.1. IntroductionSystemic lupus erythematosus (SLE) is a multi-systemic autoimmune disease of unknown etiology with heterogeneous clinical manifestations, including diverse neuropsychiatric manifestations
autoimmune disease 21712 neuroinflammatory process of CNS diseases.Recent evidence has implicated MMP-9 in the pathogenesis of a variety of autoimmune disease s [[64]]. Peripheral blood mononuclear cells (PBMCs) from SLE patients have been shown to express higher
systemic lupus erythematosus 1011 patients, disease involving the central nervous system (CNS), collectively termed neuropsychiatric systemic lupus erythematosus (NPSLE), remains one of the important causes of death in these patients. Cognitive dysfunction is one
systemic lupus erythematosus 17520 1Table 1Human studies on the association between anti-NR2A/B antibodies and cognitive dysfunction in systemic lupus erythematosus (SLE) patients.ReferencesStudy TypeNumber of SubjectsAmino Acid Sequence Used for ImmunoassaysReported

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