The Immunologic Role of Gut Microbiota in Patients with Chronic HBV Infection

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hepatitis B 7 infectiousdiseases
hepatitis C 1 infectiousdiseases
infectious disease 2 infectiousdiseases

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hepatitis B 543 /2018Publication date (epub): 7/2018AbstractHepatitis B can cause acute or chronic liver damage due to hepatitis B virus (HBV) infection. Cirrhosis or hepatocellular carcinoma (HCC) caused by chronic HBV infection often
hepatitis B 1382 discuss the immunologic role of the gut microbiota in chronic HBV infection.1. IntroductionChronic hepatitis B virus (HBV) infection is a global problem that threatens human health. Chronic HBV infection can easily
hepatitis B 1972 people die due to complications caused by chronic HBV infection [[1]]. Although the application of the hepatitis B vaccine and antiviral drugs has caused the rate of HBV infection to decrease yearly, chronic HBV infection
hepatitis B 2648 gut microbiota has been associated with alcoholic liver disease, autoimmune liver disease, chronic hepatitis B (CHB), cirrhosis and HCC; moreover, changes in the intestinal flora are dependent on the various pathogeneses
hepatitis B 2893 [[4]–[8]]. Furthermore, scholars from Taiwan have indicated that the gut microbiota may determine acute hepatitis B and CHB attacks. Specifically, the composition of the gut microbiota affects the host immune response
hepatitis B 4549 the host immune response caused by HBV infection exerts a significant influence on the prognosis of hepatitis B and the treatment effect of antiviral drugs [[12]]. The immune response against HBV mainly includes
hepatitis B 9870 Subsequently, the same changes were found in the intestinal flora of chronic HBV carriers, CHB patients, and hepatitis B -induced cirrhosis patients; that is, the structures and abundances of the bacterial groups were obviously
hepatitis C 19355 improve hepatic encephalopathy for a 57-year-old male with cirrhosis secondary to both alcohol and the hepatitis C virus [[50]]. A report on HBeAg-positive CHB therapy in patients with ongoing ETV/TDF therapy showed
infectious disease 15016 response. The CpG-TLR9 signaling pathway plays an important role in the prevention and treatment of infectious disease s [[40]]. Researches have highlighted that CpG-TLR9 and MyD88 form compounds (MyD88, IRAK4, and TRAF6),
infectious disease 17968 that the application of synthetic unmethylated CpG DNA is a promising measure for the treatment of infectious disease s [[41]]. Additionally, the physiological indexes (BMI) and serum metabolites of the patients with chronic

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