Synergistic Integration of Laboratory and Numerical Approaches in Studies of the Biomechanics of Diseased Red Blood Cells

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diabetes mellitus 1 endocrinologydiseases
diabetic retinopathy 1 endocrinologydiseases
hyperglycemia 2 endocrinologydiseases

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diabetes mellitus 51354 representative model for normal RBCs, while D-RBC1, D-RBC2, and D-RBC3 are three potential models for diabetes mellitus (DM) RBCs. The simulation results of N-RBC is compared with the experimental data [[153]] drawn in red
diabetic retinopathy 32303 tweezer stretching technique. They also showed a great decrease in the DIs of RBCs from patients with diabetic retinopathy . Moreover, numerous AFM studies have directly pointed out an evident stiffness increase in diabetic
hyperglycemia 30780 recognized as the world’s fastest growing chronic disease, is a metabolic dysfunction of the persistent hyperglycemia caused by either insulin insufficiency or insulin resistance at many body cells [[141]]. T2DM is the
hyperglycemia 32565 Young’s modulus [[32],[147],[148]]. This could be due to the glycosylation of the RBC membrane during hyperglycemia which, in turn, leads to alterations in the membrane lipid-protein interactions associated with the

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