Wet beriberi with multiple organ failure remarkably reversed by thiamine administration: A case report and literature review.

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thyrotoxicosis 1 endocrinologydiseases
beriberi 51 endocrinologydiseases
glycogen storage disease 1 endocrinologydiseases
lactic acidosis 4 endocrinologydiseases

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beriberi 19 Title: MedicineWet beriberi with multiple organ failure remarkably reversed by thiamine administrationA case report and literature
beriberi 821 particular pattern that should suggest thiamine (vitamin B1) deficiency. The clinical picture of wet beriberi secondary to thiamine deficiency only demonstrates non-specific clinical manifestations. For a diagnosis
beriberi 942 thiamine deficiency only demonstrates non-specific clinical manifestations. For a diagnosis of wet beriberi , medical history is very important. Interestingly, imprisonment was also found to be related to thiamine
beriberi 1109 imprisonment was also found to be related to thiamine deficiency. This article presents a rare case of wet beriberi associated with multiple organ failure (MOF) in a prison patient with years of heavy alcohol consumption.Patient
beriberi 2145 function improvement within a few days, which were normal within a month.Lessons:A diagnosis of wet beriberi should be considered for a prison patient who has unexplained heart failure, lactic acidosis, and/or
beriberi 2365 Moreover, the patient should be empirically given thiamine administration without delay.Introduction1Wet beriberi , secondary to thiamine (vitamin B1) deficiency, is characterized by cardiovascular damage, and it is
beriberi 2494 thiamine (vitamin B1) deficiency, is characterized by cardiovascular damage, and it is different from dry beriberi , which predominantly shows neurological involvement. We found that the vast majority of wet beriberi
beriberi 2595 beriberi, which predominantly shows neurological involvement. We found that the vast majority of wet beriberi patients have causal factors, which is key for diagnosis. Here, we reported a case of a 39-year-old
beriberi 2717 causal factors, which is key for diagnosis. Here, we reported a case of a 39-year-old man with wet beriberi who had a history of long-term drinking, imprisonment, and furosemide administration and presented with
beriberi 2999 were remarkably reversed by thiamine administration. Importantly, we reviewed the literature on wet beriberi and summarized the etiology, reports of cardiac output (CO) and systemic vascular resistance (SVR),
beriberi 3184 vascular resistance (SVR), currently available diagnostic tests, and thiamine use in patients with wet beriberi . We believe the relevant content may have important clinical implications.Case presentation2A 39-year-old
beriberi 7289 Scr = serum creatinine, SVR = systemic vascular resistance, UV = urine volume.Discussion3Wet beriberi is one of the clinical syndromes associated with thiamine deficiency. Thiamine, in its phosphorylated
beriberi 8045 Increased preload and myocardial dysfunction ultimately leads to congestive heart failure.[[2],[3]] Wet beriberi mainly triggers right heart failure.[[4]–[6]] The moderate pulmonary hypertension is common for wet
beriberi 8156 mainly triggers right heart failure.[[4]–[6]] The moderate pulmonary hypertension is common for wet beriberi patient.[[3],[4],[7]] Our patient had a pulmonary hypertension of 57 mmHg. Increased pulmonary arterial
beriberi 8575 hypertension.[[3],[4]] Hyperkinetic LVEF is observed in many patients. Attas et al[[2]] reported that the LVEF of a wet beriberi patient was 72%; Yamamura et al[[3]] reported the LVEF was 85%; our patient had a LVEF of 77.1%. Certain
beriberi 9205 acidosis, in the setting of alcoholism, chronic malnutrition, and so on.[[1]]Based on the standard for wet beriberi ,[[2]] wet beriberi was established in our patient. Besides this, wet beriberi has a rare and severe
beriberi 9224 of alcoholism, chronic malnutrition, and so on.[[1]]Based on the standard for wet beriberi,[[2]] wet beriberi was established in our patient. Besides this, wet beriberi has a rare and severe form; Shoshin beriberi
beriberi 9283 the standard for wet beriberi,[[2]] wet beriberi was established in our patient. Besides this, wet beriberi has a rare and severe form; Shoshin beriberi is an acute and fulminant form of wet beriberi, which is
beriberi 9328 beriberi was established in our patient. Besides this, wet beriberi has a rare and severe form; Shoshin beriberi is an acute and fulminant form of wet beriberi, which is described as a “rapidly curable hemodynamic
beriberi 9375 this, wet beriberi has a rare and severe form; Shoshin beriberi is an acute and fulminant form of wet beriberi , which is described as a “rapidly curable hemodynamic disaster” that is characterized by hypotension,
beriberi 9811 serum thiamine concentration was low. These further confirmed that the patient was diagnosed with wet beriberi .For a diagnosis of wet beriberi, the patient's medical history is very important. The etiology of wet
beriberi 9843 low. These further confirmed that the patient was diagnosed with wet beriberi.For a diagnosis of wet beriberi , the patient's medical history is very important. The etiology of wet beriberi is illustrated in Fig.
beriberi 9922 beriberi.For a diagnosis of wet beriberi, the patient's medical history is very important. The etiology of wet beriberi is illustrated in Fig. 2.[[1]–[5],[7]–[25]] Long-term drinking can lead to decreased vitamin B1
beriberi 10157 dysfunction and can increase the amount of damage; long-term drinking was the most common cause of wet beriberi . According to whether the patient has a history of long-term drinking or not, beriberi can be divided
beriberi 10244 cause of wet beriberi. According to whether the patient has a history of long-term drinking or not, beriberi can be divided into alcohol-related beriberi and non-alcohol-related beriberi. Digestive system diseases
beriberi 10289 the patient has a history of long-term drinking or not, beriberi can be divided into alcohol-related beriberi and non-alcohol-related beriberi. Digestive system diseases and surgery were the other major causes
beriberi 10322 long-term drinking or not, beriberi can be divided into alcohol-related beriberi and non-alcohol-related beriberi . Digestive system diseases and surgery were the other major causes of thiamine deficiency. Notably,
beriberi 10736 imprisonment was also found to be related to thiamine deficiency. Zhang et al[[15]] reported 3 cases of wet beriberi in prison and mentioned that all of the prisoners had various symptoms of thiamine deficiency. Park
beriberi 10877 of the prisoners had various symptoms of thiamine deficiency. Park et al[[4]] also reported on wet beriberi in prisons in western society. Cisse et al[[27]] studied Guinean prisons from 2010 to 2014 and found
beriberi 10999 western society. Cisse et al[[27]] studied Guinean prisons from 2010 to 2014 and found 38 cases of beriberi secondary to thiamine deficiency. There were 14 cases of demonstrated wet beriberi and 2 cases of Shoshin
beriberi 11082 found 38 cases of beriberi secondary to thiamine deficiency. There were 14 cases of demonstrated wet beriberi and 2 cases of Shoshin beriberi. Heart failure was a common observation in patients with wet beriberi.
beriberi 11114 secondary to thiamine deficiency. There were 14 cases of demonstrated wet beriberi and 2 cases of Shoshin beriberi . Heart failure was a common observation in patients with wet beriberi. Furosemide is a loop diuretic
beriberi 11184 beriberi and 2 cases of Shoshin beriberi. Heart failure was a common observation in patients with wet beriberi . Furosemide is a loop diuretic that increases urinary output, thereby reducing edema in patients with
beriberi 11718 presented with thiamine deficiency that resulted from furosemide administration.[[29]]Figure 2Etiology of beriberi . From 24 documents including 31 cases. Long-term drinking, digestive system disease and surgery, imprisonment,
beriberi 12255 document.Even though thiamine deficiency is a non-uncommon situation in critically ill patients, wet beriberi is easily misdiagnosed and missed diagnosis. First, the typical features of wet beriberi are high CO
beriberi 12344 patients, wet beriberi is easily misdiagnosed and missed diagnosis. First, the typical features of wet beriberi are high CO and low SVR, but the typical features are often absent in many patients (Table 3[[2],[3],[5],[7]–[9],[17]–[19]]).
beriberi 12941 hypertension.[[4]] MOF is not uncommon.[[19],[20]] Acute renal failure was the most common complication of wet beriberi , with some of the patients requiring CRRT.[[2],[5],[8]–[10],[14],[16],[18]–[22]] Watson et al[[30]]
beriberi 13089 CRRT.[[2],[5],[8]–[10],[14],[16],[18]–[22]] Watson et al[[30]] reported that 39.4% (13/33) of wet beriberi patients had acute renal failure with high levels of blood lactate and pyruvate, which produced peripheral
beriberi 13397 decreased glomerular filtration rate. Acute liver failure is another complication associated with wet beriberi .[[14],[16],[19],[20]] Watson et al[[30]] reported that 18.8% (6/32) of wet beriberi patients had acute
beriberi 13481 associated with wet beriberi.[[14],[16],[19],[20]] Watson et al[[30]] reported that 18.8% (6/32) of wet beriberi patients had acute liver failure related to hepatic congestion caused by right heart failure. In addition
beriberi 13980 especially the measurement of thiamine concentration, in documents discussing the diagnosis of wet beriberi (Table 4). Notably, there was lack of sensitivity and specificity data on these tests.Table 3Reports
beriberi 14193 cardiac output and systemic vascular resistance.Table 4Currently available diagnostic tests for wet beriberi .Treatment with vitamin B1 is useful for the diagnosis and treatment of wet beriberi. It is generally
beriberi 14277 diagnostic tests for wet beriberi.Treatment with vitamin B1 is useful for the diagnosis and treatment of wet beriberi . It is generally accepted that suspected patients should be given a therapeutic administration of thiamine.
beriberi 14956 reaction) and 11 cases of transient local irritation (minor reaction).Administration of thiamine for wet beriberi varies among authors (Table 5). Generally, alcohol-related beriberi patients were administered a higher
beriberi 15024 reaction).Administration of thiamine for wet beriberi varies among authors (Table 5). Generally, alcohol-related beriberi patients were administered a higher dose of thiamine than non-alcohol-related beriberi patients, because
beriberi 15111 alcohol-related beriberi patients were administered a higher dose of thiamine than non-alcohol-related beriberi patients, because alcohol can inhibit the uptake of vitamin B1 and the phosphorylation of its active
beriberi 15272 vitamin B1 and the phosphorylation of its active form (TPP).[[12],[32]] For non-alcohol-related wet beriberi patients, currently, the most common treatment is a daily intravenous treatment of 100 to 200 mg of
beriberi 15569 of life-threatening conditions;[[12]] later, oral administration is indicated. For alcohol-related beriberi patients, doses should be increased appropriately.Table 5Reports of thiamine use.For Shoshin beriberi
beriberi 15671 beriberi patients, doses should be increased appropriately.Table 5Reports of thiamine use.For Shoshin beriberi patients, after thiamine treatment, the hemodynamic indices (blood pressure, pulse, urine volume, serum
beriberi 16229 stopped vasopressor treatment in the following 48 hours.Conclusion4We have presented a case of wet beriberi (Shoshin beriberi) associated with MOF in a prison patient with a history of heavy alcohol consumption,
beriberi 16247 treatment in the following 48 hours.Conclusion4We have presented a case of wet beriberi (Shoshin beriberi ) associated with MOF in a prison patient with a history of heavy alcohol consumption, an especially
glycogen storage disease 12013 factors mean >1 factor, and at least include one of the factors mentioned above. Other cause was type-1 glycogen storage disease , which was reported in 1 case, and there was 1 case with unknown cause because the author did not mention
lactic acidosis 2231 diagnosis of wet beriberi should be considered for a prison patient who has unexplained heart failure, lactic acidosis , and/or MOF. Moreover, the patient should be empirically given thiamine administration without delay.Introduction1Wet
lactic acidosis 9091 days.[[11]–[14]] Thiamine deficiency should be suspected in patients with unexplained heart failure and lactic acidosis , in the setting of alcoholism, chronic malnutrition, and so on.[[1]]Based on the standard for wet beriberi,[[2]]
lactic acidosis 9507 a “rapidly curable hemodynamic disaster” that is characterized by hypotension, tachycardia, and lactic acidosis .[[1],[5],[7],[8],[11],[12]] The patient's condition improved after thiamine administration, vasopressor
lactic acidosis 16590 forgotten but memorable disease. Patients with malnourished diet who have unexplained heart failure, lactic acidosis , and/or MOF should be empirically given thiamine administration without delay.Supplementary MaterialSupplemental
thyrotoxicosis 687 textAbstractAbstractRationale:Circulatory failure, especially with low systemic vascular resistance (SVR), as observed in septic shock, thyrotoxicosis , and anemia, is a particular pattern that should suggest thiamine (vitamin B1) deficiency. The clinical

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