Association of osteoarthritis and circulating adiponectin levels: a systematic review and meta-analysis

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obesity 7 endocrinologydiseases

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obesity 2422 suggested that OA is a systemic disorder with a multifactorial origin. The systemic risk factors include obesity , gender, injury, age and a genetic bias [[2]]. Moreover, there are increasing evidences indicated that
obesity 2533 gender, injury, age and a genetic bias [[2]]. Moreover, there are increasing evidences indicated that obesity is an essential element in the pathogenesis of OA [[7]]. Sowers et al. [[8]] have suggested that the
obesity 2661 element in the pathogenesis of OA [[7]]. Sowers et al. [[8]] have suggested that the mechanism by which obesity is involved in OA, may be a simple increase under the mechanical burden in the joints. However, Gabay
obesity 2803 increase under the mechanical burden in the joints. However, Gabay et al. [[9]] indicated that the obesity induced high metabolic and inflammatory environments play crucial roles in the onset of OA. What’s
obesity 3369 tissue proteins but also is the only adipokine identified thus far that is negatively correlated with obesity [[15]]. For many years, the effects of adiponectin in many metabolic conditions such as insulin resistance,
obesity 18702 strategies in hand or hip OA patient. As for knee joint, one of weight-bearing joint most influenced by obesity , was confirmed to be more susceptibility through the action of inflammatory adipokines than hip joint
obesity 19102 adiponectin was secreted only from adipose tissue. Which suggested a more complex relationship among obesity , adiponectin and OA and might further approve of our hypothesis that adiponectin increase compensatorily

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