Elucidating Adverse Nutritional Implications of Exposure to Endocrine-Disrupting Chemicals and Mycotoxins through Stable Isotope Techniques.

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Annotation Summary

Term Occurence Count Dictionary
Insulin 1 endocrinologydiseasesdrugs
ethinylestradiol 1 endocrinologydiseasesdrugs
hypothyroidism 1 endocrinologydiseases
kwashiorkor 1 endocrinologydiseases
metabolic syndrome 1 endocrinologydiseases
obesity 10 endocrinologydiseases

Graph of close proximity drug and disease terms (within 200 characters).

Note: If this graph is empty, then there are no terms that meet the proximity constraint.

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Select Drug Character Offset Drug Term Instance
Insulin 15619 resistant and that poor intrauterine growth predicted higher central adiposity at 8 years of age [[43]]. Insulin resistance associated with intrauterine growth restriction was linkedwith postanatal catch-up growth
ethinylestradiol 9181 manufacturing processes or emerge as breakdown products of other chemical compounds. Some, such as ethinylestradiol and diethylstilbestrol, are synthetic human or animal drugs, while others are found in nature, such
Select Disease Character Offset Disease Term Instance
hypothyroidism 12796 excessively tall or abnormally short [[31]]. Furthermore, exposure to environmental chemicals may result in hypothyroidism , a condition related to very low levels of thyroid hormones in the blood, which may lead to slower growth
kwashiorkor 17001 and breast milk [[44],[51]]. Aflatoxin exposure has been linked to impaired growth [[14],[22]] and kwashiorkor [[50]], and may also have a role in the modification of the aetiology of hepatitis B [[52],[53]] in
metabolic syndrome 8486 include abnormal growth patterns, male and female reproductive abnormalities, neurobehavioral deficits, metabolic syndrome , bone disorders, immune disorders and some types of cancer [[12]].EDCs represent a broad class of molecules
obesity 757 energy-dense foods based on meats, milk, animal fats and vegetable oils. The shift to overweight and obesity is driven by increased exposure to mass media, urbanization, technological advances in food processing,
obesity 1638 disrupt the endocrine system. These hazards sit at the cross road of undernutrition and overweight and obesity since the exposure cuts across the critical window of opportunity (the first 1000 days). In this review,
obesity 2489 co-existence of chronic undernutrition and micronutrient deficiencies on the one hand and overweight and obesity on the other. The double burden of malnutrition (DBM) affects at least 40% of countries worldwide [[1]].
obesity 2864 from overweight [[1]]. Growth retardation in the first 1000 days is linked to risk for overweight, obesity and related non-communicable diseases (NCDs) in later childhood, adolescence and adulthood [[2]]. All
obesity 3945 of between US$1005 and US$3955 or less in 2016 [[5]]. Major drivers of the shift to overweight and obesity in LMICs have been identified as increased exposure to mass media due to globalisation, urbanization,
obesity 5096 endocrine system, amongst others. These hazards sit at the cross road of undernutrition and overweight and obesity since the exposure cuts across the critical window of opportunity (the first 1000 days) from pregnancy
obesity 8103 then leads to metabolic programming favouring excess adiposity eventually resulting in overweight and obesity . Changes in microbiota composition and function and gene alterations may also be involved but more research
obesity 14249 of hormones and cytokines [[37]]. These metabolic changes are likely to increase pre-disposition to obesity .Obesogens, including EDCs, may interfere with critical metabolic signalling, for example nuclear hormone
obesity 15074 thought to be due to an early onset of adiposity rebound. A recent review of the link between EDCs and obesity observed that prenatal exposure to some of the chemicals lead to decreased birth weight which is in
obesity 22288 fat accumulation and distribution is an important indicator for assessing the risk for overweight, obesity and related NCDs. Excess body fat is associated with physiological changes that can lead to NCDs, such

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